Search results for "Single unit electrophysiology"

showing 2 items of 2 documents

Nitric oxide-active compounds modulate the intensity of glutamate-evoked responses in the globus pallidus of the rat

2011

Abstract Aim The effects of local applied NO-active compounds on glutamate (GLU)-evoked responses were investigated in globus pallidus (GP) neurons. Main methods Extracellularly recorded single units from anesthetized rats were treated with GLU before and during the microiontophoretic application of S-nitrosoglutathione (SNOG), a NO donor, and Nω-nitro- l -arginine methyl ester (L-NAME), a NOS inhibitor. Key findings Most GP cells were excited by SNOG whereas administration of L-NAME induced decrease of GP neurons activity. Nearly all neurons responding to SNOG and/or L-NAME showed significant modulation of their excitatory responses to the administration of iontophoretic GLU. In these cell…

MaleNOS inhibitorGlutamic AcidNitric oxide - Microiontophoresis - ElectrophysiologyBiologyPharmacologyGlobus PallidusNitric OxideSettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular BiologyNitric oxidechemistry.chemical_compoundGlutamatergicNitric oxide; Basal ganglia; Single unit electrophysiology; MicroiontophoresisBasal gangliaSingle unit electrophysiologyAnimalsNitric Oxide DonorsRats WistarGeneral Pharmacology Toxicology and PharmaceuticsEvoked PotentialsNeuronsMicroiontophoresisIontophoresisGlutamate receptorExcitatory Postsynaptic PotentialsGeneral MedicineIontophoresisRatsNG-Nitroarginine Methyl EsterGlobus pallidusBiochemistrychemistryBasal gangliaExcitatory postsynaptic potentialNitric Oxide SynthaseMicroelectrodesLife Sciences
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Nitric oxide- and cGMP-active compounds affect the discharge of substantia nigra pars reticulata neurons: in vivo evidences in the rat

2009

The nitric oxide (NO)-active drugs influence on the bioelectric activity of neurons of the pars reticulata of the substantia nigra was studied in urethane-anesthetized rats. A first group of animals was treated with 7-nitro-indazole (7-NI), a preferential inhibitor of neuronal NO synthase. In a second group of rats, electrophysiological recordings were coupled with microiontophoretic administration of Nomega-nitro-L-arginine methyl ester (L-NAME, a NO synthase inhibitor), 3-morpholino-sydnonimin-hydrocloride (SIN-1, a NO donor) and 8-Br-cGMP (a cell-permeable analogue of cGMP, the main second-messenger of NO neurotransmission). 7-NI and L-NAME caused a statistically significant decrease in …

MaleSubstantia nigra pars reticulataAction PotentialsDown-RegulationSubstantia nigraNitric Oxide Synthase Type INeurotransmissionPharmacologyBiologySettore BIO/09 - FisiologiaNitric oxidechemistry.chemical_compoundIn vivoAnimalsSingle unit electrophysiologyNitric Oxide DonorsEnzyme InhibitorsRats WistarCyclic GMPBiological PsychiatrySubstantia nigra pars reticulataNeuronsMicroiontophoresisNeural InhibitionNitric oxideIontophoresisRatsUp-RegulationSubstantia NigraPsychiatry and Mental healthElectrophysiologyNG-Nitroarginine Methyl EsterNeurologychemistryMolsidomineExcitatory postsynaptic potentialNeurology (clinical)Pars reticulataNeuroscienceSignal TransductionJournal of Neural Transmission
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